faculty photo

 

Robert A. McKnight, Ph.D.
Assistant Professor

Education

  • Iowa State University - BS Biology
  • University of Southwestern Louisiana - MS Microbiology
  • University of California, Davis - PhD Genetics

Current Appointments

  • Research Assistant Professor - University of Utah, Department of Pediatrics

More Recent Publications [Search PubMed for Additional Publications]

  • Nonresponsiveness of cerebral p53-MDM2 functional circuit in newborn rat pups rendered IUGR via uteroplacental insufficiency.
        Ke X, McKnight RA, Wang ZM, Yu X, Wang L, Callaway CW, Albertine KH, Lane RH. Am J Physiol Regul Integr Comp Physiol. 2005
  • Uteroplacental insufficiency induces site-specific changes in histone H3 covalent modifications and affects DNA-histone H3 positioning in day 0 IUGR rat liver.
        Fu Q, McKnight RA, Yu X, Wang L, Callaway CW, Lane RH. Physiol Genomics. 2004
  • Glucose transporter GLUT8 translocation in neurons is not insulin responsive.
        Shin BC, McKnight RA, Devaskar SU. J Neurosci Res.
  • Aberrant insulin-induced GLUT4 translocation predicts glucose intolerance in the offspring of a diabetic mother.
        Thamotharan M, McKnight RA, Thamotharan S, Kao DJ, Devaskar SU. Am. J. Physiol. Endocrinol. Metab.
  • GLUT8 is a glucose transporter responsible for insulin-stimulated glucose uptake in the blastocyst.
        Carayannopoulous MO, Chi MMY, Cui Y, Pingsterhaus JM, McKnight RA, et.al. P.N.A.S.